Regression of left ventricular mass in hypertensive patients treated with perindopril/indapamide as a first-line combination: The REASON echocardiography study
Identifieur interne : 00AB12 ( Main/Exploration ); précédent : 00AB11; suivant : 00AB13Regression of left ventricular mass in hypertensive patients treated with perindopril/indapamide as a first-line combination: The REASON echocardiography study
Auteurs : Nicola De Luca [Italie] ; Jean-Michel Mallion [France] ; Michael F. O'Rourke [Australie] ; Eoin O'Brien [Irlande (pays)] ; Karl-Heinz Rahn [Allemagne] ; Bruno Trimarco [Italie] ; Ramon Romero [Espagne] ; Peter Wilhelmus De Leeuw [Pays-Bas] ; Gerhart Hitzenberger [Autriche] ; Edouard Battegay [Suisse] ; Daniel Duprez [Belgique] ; Peter Sever [Royaume-Uni] ; Michel E. Safar [France]Source :
- American journal of hypertension [ 0895-7061 ] ; 2004.
Descripteurs français
- Pascal (Inist)
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- topic : Homme.
English descriptors
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Abstract
Background: Increase in left ventricular mass (LVM) may be linked to morbidity and mortality in hypertensive patients. Arterial stiffness, systolic blood pressure (BP), and pulse pressure (PP) seem to be the main determinants of LVM. The perindopril/indapamide combination normalizes systolic BP, PP, and arterial function to a greater extent than atenolol. The aim of this study was to compare the effects of perindopril (2 mg)/indapamide (0.625 mg) first-line combination with atenolol (50 mg) on LVM reduction in hypertensive patients. Methods: Two hundred fourteen patients with essential hypertension participating in the PREterax in Regression of Arterial Stiffness in a Controlled Double-BliNd (REASON), randomized, double-blind, parallel-group study, underwent M-mode two-dimensional-guided echocardiography. Results: Perindopril/indapamide and atenolol were both effective at brachial BP reduction during the 12-month period. The systolic BP reduction was significantly greater with perindopril/indapamide than with atenolol (-21.2 v -15.3 mm Hg), whereas the reduction in diastolic BP was similar between treatment groups (-12.1 v -11.3 mm Hg). Reduction in LVM was higher with perindopril/indapamide than with atenolol. The between-group difference was significant for LVM (-13.6 v -4.3 g, P = .027), LVM/body surface area (LVMI1, P = .032), and LVM/body height2.7 (LVMI2, P = .013). The 124 patients with LV hypertrophy at baseline showed greatest LVM regression (LVM: -22.5 v -8.9 g, P = .009; LVMI 1, P = .031; LVMI2, P = .028). The reduction in LVM adjusted for brachial systolic BP and heart rate was still significantly greater with perindopril/indapamide than with atenolol. Conclusions: Treatment, based on a first-line perindopril/indapamide combination in hypertensive patients, was more effective than atenolol on regression of echocardiographic indices of LVM and LV hypertrophy.
Affiliations:
- Allemagne, Australie, Autriche, Belgique, Espagne, France, Irlande (pays), Italie, Pays-Bas, Royaume-Uni, Suisse
- Angleterre, District de Münster, Grand Londres, Rhénanie-du-Nord-Westphalie, Vienne (Autriche), Île-de-France
- Londres, Münster, Paris, Vienne (Autriche)
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O'Rourke</name><affiliation wicri:level="1"><inist:fA14 i1="03"><s1>Saint Vincent's Hospital</s1><s2>Darlinghurst</s2><s3>AUS</s3><sZ>3 aut.</sZ></inist:fA14><country>Australie</country><wicri:noRegion>Saint Vincent's Hospital</wicri:noRegion></affiliation></author><author><name sortKey="O Brien, Eoin" sort="O Brien, Eoin" uniqKey="O Brien E" first="Eoin" last="O'Brien">Eoin O'Brien</name><affiliation wicri:level="1"><inist:fA14 i1="04"><s1>Department of Clinical Pharmacology, Royal College of Surgeons in Ireland, and Blood Pressure Unit, Beaumont Hospital</s1><s2>Dublin</s2><s3>IRL</s3><sZ>4 aut.</sZ></inist:fA14><country>Irlande (pays)</country><wicri:noRegion>Dublin</wicri:noRegion></affiliation></author><author><name sortKey="Rahn, Karl Heinz" sort="Rahn, Karl Heinz" uniqKey="Rahn K" first="Karl-Heinz" last="Rahn">Karl-Heinz Rahn</name><affiliation wicri:level="3"><inist:fA14 i1="05"><s1>Medizinische Poliklinik. University of Münster</s1><s2>Münster</s2><s3>DEU</s3><sZ>5 aut.</sZ></inist:fA14><country>Allemagne</country><placeName><region type="land" nuts="1">Rhénanie-du-Nord-Westphalie</region><region type="district" nuts="2">District de Münster</region><settlement type="city">Münster</settlement></placeName></affiliation></author><author><name sortKey="Trimarco, Bruno" sort="Trimarco, Bruno" uniqKey="Trimarco B" first="Bruno" last="Trimarco">Bruno Trimarco</name><affiliation wicri:level="1"><inist:fA14 i1="01"><s1>Clinica Medica</s1><s2>Napoli</s2><s3>ITA</s3><sZ>1 aut.</sZ><sZ>6 aut.</sZ></inist:fA14><country>Italie</country><wicri:noRegion>Clinica Medica</wicri:noRegion></affiliation></author><author><name sortKey="Romero, Ramon" sort="Romero, Ramon" uniqKey="Romero R" first="Ramon" last="Romero">Ramon Romero</name><affiliation wicri:level="1"><inist:fA14 i1="06"><s1>Hospital Germans Trias y Pujol</s1><s2>Badalona</s2><s3>ESP</s3><sZ>7 aut.</sZ></inist:fA14><country>Espagne</country><wicri:noRegion>Hospital Germans Trias y Pujol</wicri:noRegion></affiliation></author><author><name sortKey="De Leeuw, Peter Wilhelmus" sort="De Leeuw, Peter Wilhelmus" uniqKey="De Leeuw P" first="Peter Wilhelmus" last="De Leeuw">Peter Wilhelmus De Leeuw</name><affiliation wicri:level="1"><inist:fA14 i1="07"><s1>Academisch Ziekenhuis Maastricht</s1><s2>Maastricht</s2><s3>NLD</s3><sZ>8 aut.</sZ></inist:fA14><country>Pays-Bas</country><wicri:noRegion>Academisch Ziekenhuis Maastricht</wicri:noRegion></affiliation></author><author><name sortKey="Hitzenberger, Gerhart" sort="Hitzenberger, Gerhart" uniqKey="Hitzenberger G" first="Gerhart" last="Hitzenberger">Gerhart Hitzenberger</name><affiliation wicri:level="3"><inist:fA14 i1="08"><s1>Institut für Hypertoniker</s1><s2>Wien</s2><s3>AUT</s3><sZ>9 aut.</sZ></inist:fA14><country>Autriche</country><placeName><settlement type="city">Vienne (Autriche)</settlement><region nuts="2" type="province">Vienne (Autriche)</region></placeName></affiliation></author><author><name sortKey="Battegay, Edouard" sort="Battegay, Edouard" uniqKey="Battegay E" first="Edouard" last="Battegay">Edouard Battegay</name><affiliation wicri:level="1"><inist:fA14 i1="09"><s1>Medizinische Universitäts Poliklinik</s1><s2>Basel</s2><s3>CHE</s3><sZ>10 aut.</sZ></inist:fA14><country>Suisse</country><wicri:noRegion>Medizinische Universitäts Poliklinik</wicri:noRegion></affiliation></author><author><name sortKey="Duprez, Daniel" sort="Duprez, Daniel" uniqKey="Duprez D" first="Daniel" last="Duprez">Daniel Duprez</name><affiliation wicri:level="1"><inist:fA14 i1="10"><s1>Universitair Ziekenhuis Gent</s1><s2>Gent</s2><s3>BEL</s3><sZ>11 aut.</sZ></inist:fA14><country>Belgique</country><wicri:noRegion>Universitair Ziekenhuis Gent</wicri:noRegion></affiliation></author><author><name sortKey="Sever, Peter" sort="Sever, Peter" uniqKey="Sever P" first="Peter" last="Sever">Peter Sever</name><affiliation wicri:level="3"><inist:fA14 i1="11"><s1>Saint Mary's Hospital Medical School</s1><s2>London</s2><s3>GBR</s3><sZ>12 aut.</sZ></inist:fA14><country>Royaume-Uni</country><placeName><settlement type="city">Londres</settlement><region type="country">Angleterre</region><region type="région" nuts="1">Grand Londres</region></placeName></affiliation></author><author><name sortKey="Safar, Michel E" sort="Safar, Michel E" uniqKey="Safar M" first="Michel E." last="Safar">Michel E. Safar</name><affiliation wicri:level="3"><inist:fA14 i1="12"><s1>Hôpital Broussais</s1><s2>Paris</s2><s3>FRA</s3><sZ>13 aut.</sZ></inist:fA14><country>France</country><placeName><region type="region">Île-de-France</region><region type="old region">Île-de-France</region><settlement type="city">Paris</settlement></placeName></affiliation></author></analytic><series><title level="j" type="main">American journal of hypertension</title><title level="j" type="abbreviated">Am. j. hypertens.</title><idno type="ISSN">0895-7061</idno><imprint><date when="2004">2004</date></imprint></series></biblStruct></sourceDesc><seriesStmt><title level="j" type="main">American journal of hypertension</title><title level="j" type="abbreviated">Am. j. hypertens.</title><idno type="ISSN">0895-7061</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Cardiovascular disease</term><term>Echocardiography</term><term>Human</term><term>Hypertension</term><term>Indapamide</term><term>Left ventricle</term><term>Perindopril</term><term>Treatment</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Hypertension artérielle</term><term>Ventricule gauche</term><term>Homme</term><term>Appareil circulatoire pathologie</term><term>Traitement</term><term>Périndopril</term><term>Indapamide</term><term>Echocardiographie</term></keywords><keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Homme</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Background: Increase in left ventricular mass (LVM) may be linked to morbidity and mortality in hypertensive patients. Arterial stiffness, systolic blood pressure (BP), and pulse pressure (PP) seem to be the main determinants of LVM. The perindopril/indapamide combination normalizes systolic BP, PP, and arterial function to a greater extent than atenolol. The aim of this study was to compare the effects of perindopril (2 mg)/indapamide (0.625 mg) first-line combination with atenolol (50 mg) on LVM reduction in hypertensive patients. Methods: Two hundred fourteen patients with essential hypertension participating in the PREterax in Regression of Arterial Stiffness in a Controlled Double-BliNd (REASON), randomized, double-blind, parallel-group study, underwent M-mode two-dimensional-guided echocardiography. Results: Perindopril/indapamide and atenolol were both effective at brachial BP reduction during the 12-month period. The systolic BP reduction was significantly greater with perindopril/indapamide than with atenolol (-21.2 v -15.3 mm Hg), whereas the reduction in diastolic BP was similar between treatment groups (-12.1 v -11.3 mm Hg). Reduction in LVM was higher with perindopril/indapamide than with atenolol. The between-group difference was significant for LVM (-13.6 v -4.3 g, P = .027), LVM/body surface area (LVMI<sub>1</sub>, P = .032), and LVM/body height<sup>2.7</sup> (LVMI<sub>2</sub>, P = .013). The 124 patients with LV hypertrophy at baseline showed greatest LVM regression (LVM: -22.5 v -8.9 g, P = .009; LVMI <sub>1</sub>, P = .031; LVMI<sub>2</sub>, P = .028). The reduction in LVM adjusted for brachial systolic BP and heart rate was still significantly greater with perindopril/indapamide than with atenolol. Conclusions: Treatment, based on a first-line perindopril/indapamide combination in hypertensive patients, was more effective than atenolol on regression of echocardiographic indices of LVM and LV hypertrophy.</div></front></TEI><affiliations><list><country><li>Allemagne</li><li>Australie</li><li>Autriche</li><li>Belgique</li><li>Espagne</li><li>France</li><li>Irlande (pays)</li><li>Italie</li><li>Pays-Bas</li><li>Royaume-Uni</li><li>Suisse</li></country><region><li>Angleterre</li><li>District de Münster</li><li>Grand Londres</li><li>Rhénanie-du-Nord-Westphalie</li><li>Vienne (Autriche)</li><li>Île-de-France</li></region><settlement><li>Londres</li><li>Münster</li><li>Paris</li><li>Vienne (Autriche)</li></settlement></list><tree><country name="Italie"><noRegion><name sortKey="De Luca, Nicola" sort="De Luca, Nicola" uniqKey="De Luca N" first="Nicola" last="De Luca">Nicola De Luca</name></noRegion><name sortKey="Trimarco, Bruno" sort="Trimarco, Bruno" uniqKey="Trimarco B" first="Bruno" last="Trimarco">Bruno Trimarco</name></country><country name="France"><noRegion><name sortKey="Mallion, Jean Michel" sort="Mallion, Jean Michel" uniqKey="Mallion J" first="Jean-Michel" last="Mallion">Jean-Michel Mallion</name></noRegion><name sortKey="Safar, Michel E" sort="Safar, Michel E" uniqKey="Safar M" first="Michel E." last="Safar">Michel E. Safar</name></country><country name="Australie"><noRegion><name sortKey="O Rourke, Michael F" sort="O Rourke, Michael F" uniqKey="O Rourke M" first="Michael F." last="O'Rourke">Michael F. O'Rourke</name></noRegion></country><country name="Irlande (pays)"><noRegion><name sortKey="O Brien, Eoin" sort="O Brien, Eoin" uniqKey="O Brien E" first="Eoin" last="O'Brien">Eoin O'Brien</name></noRegion></country><country name="Allemagne"><region name="Rhénanie-du-Nord-Westphalie"><name sortKey="Rahn, Karl Heinz" sort="Rahn, Karl Heinz" uniqKey="Rahn K" first="Karl-Heinz" last="Rahn">Karl-Heinz Rahn</name></region></country><country name="Espagne"><noRegion><name sortKey="Romero, Ramon" sort="Romero, Ramon" uniqKey="Romero R" first="Ramon" last="Romero">Ramon Romero</name></noRegion></country><country name="Pays-Bas"><noRegion><name sortKey="De Leeuw, Peter Wilhelmus" sort="De Leeuw, Peter Wilhelmus" uniqKey="De Leeuw P" first="Peter Wilhelmus" last="De Leeuw">Peter Wilhelmus De Leeuw</name></noRegion></country><country name="Autriche"><region name="Vienne (Autriche)"><name sortKey="Hitzenberger, Gerhart" sort="Hitzenberger, Gerhart" uniqKey="Hitzenberger G" first="Gerhart" last="Hitzenberger">Gerhart Hitzenberger</name></region></country><country name="Suisse"><noRegion><name sortKey="Battegay, Edouard" sort="Battegay, Edouard" uniqKey="Battegay E" first="Edouard" last="Battegay">Edouard Battegay</name></noRegion></country><country name="Belgique"><noRegion><name sortKey="Duprez, Daniel" sort="Duprez, Daniel" uniqKey="Duprez D" first="Daniel" last="Duprez">Daniel Duprez</name></noRegion></country><country name="Royaume-Uni"><region name="Angleterre"><name sortKey="Sever, Peter" sort="Sever, Peter" uniqKey="Sever P" first="Peter" last="Sever">Peter Sever</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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